Md. Habibur Rahman, PhDPostdoctoral Fellow

    125 Paterson Street, Room 7120
    New Brunswick, NJ 08901
    Email: habibur.rahman[at]


    • Post-doctoral researcher, Brain Science & Engineering Institute, Kyungpook National University, Daegu, South of Korea, September 2020 – March 2021
    • Ph.D. (Integrated Master-Doctoral course, Biomedical Science, Kyungpook National University, Daegu, South Korea, August 2020
    • Bachelor of Pharmacy, Bangladesh University, Dhaka, Bangladesh, August 2010

    Honors & Awards

    • BK21 Plus KNU Biomedical Convergence Program for Creative Talent Fellowship, Kyungpook National University, Daegu, South Korea, 2013 – 2019
    • Emerging Scientist and Best Presenter Award, BK21 Plus KNU Biomedical Convergence Program Conference, Daegu, South Korea, 2017
    • KNU Honors Scholarship for the program of integrated MS-PhD, Kyungpook National University, Daegu, South Korea, 2013 – 2017
    • University Merit Scholarship for the program of bachelor of pharmacy, Bangladesh University, Dhaka, Bangladesh, 2005 – 2009


    • Registered as a Graduate Pharmacist, Bangladesh Pharmacy Council, Dhaka, Bangladesh, 2015 –

    Short Biography

    From the beginning of my graduate-level research, I have always been interested in understanding the relationship between metabolism and immunology, and its implications in neurobiology and metabolic diseases. I obtained my bachelor’s degree in pharmacy from Bangladesh University and have recently graduated with a Ph.D. in biomedical science from Kyungpook National University (KNU), South Korea. My graduate training at KNU began by investigating the relationship between metabolic and immunological factors involved in the pathogenesis of peripheral diabetic neuropathy. I identified that a key regulatory mitochondrial enzyme, pyruvate dehydrogenase kinase (PDK), mediates a glycolytic metabolic shift in the dorsal root ganglion and has a critical role in the pathogenesis of painful diabetic neuropathy. These findings led me to investigate the role of PDK in the central nervous system following diabetes and obesity that it similarly has a  key role in metabolic reprogramming and the inflammatory activation of astrocytes.

    I joined the Buettner lab to continue my training and investigating the role of hypothalamic inflammation in disrupting brain control of metabolism and immunity.

    Selected Publications

    • Rahman MH, Bhusal A, Kim JH, Jha MK, Go Y, Jang IL, Lee IK, and Suk K. (2020). Astrocytic pyruvate dehydrogenase kinase-2 is involved in hypothalamic inflammation in mouse models of diabetes. Nature Communications. 11(1): 5906. doi: 10.1038/s41467-020-19576-1
    • Bhusal A*, Rahman MH*, Lee WH, Lee IK, and Suk K. (2020). Satellite glia as a critical component of diabetic neuropathy: role of lipocalin-2 and pyruvate dehydrogenase kinase-2 axis in the dorsal root ganglion. Glia. 2020; 1-26. doi: 10.1002/glia.23942. (*co-first authors)
    • Rahman MH, Bhusal A, Lee WH, Lee IK, and Suk K. (2018). Hypothalamic inflammation and malfunctioning glia in the pathophysiology of obesity and diabetes: Translational significance. Biochemical Pharmacology. 153, 123-133. doi: 10.1016/j.bcp.2018.01.024
    • Rahman MH, Jha MK, Kim JH, Nam Y, Lee MG, Go Y, et al. (2016) Pyruvate Dehydrogenase Kinase-mediated Glycolytic Metabolic Shift in the Dorsal Root Ganglion Drives Painful Diabetic Neuropathy. Journal of Biological Chemistry. 291(11), 6011-6025. doi: 10.1074/jbc.M115.699215.